 |
by André Voisin
CHAPTER 37
Supplementary feeding as a means of
protection against grass tetany

Rectify the faults in tetanigenic herbage at their foundation

Leaving aside the question of under-feeding, the main cause of
hypomagnesaemic tetany is known to be an imbalance among the mineral as
well as the organic elements.
The most effective, safe and simple method consists, as has been described
above, in:
| (a)
|
correcting the imbalances in the soil by applying the necessary
fertilizer;
|
|
| (b)
|
allowing the herbage to mature 1
and thus lose its tetanigenic qualities.
|
|

For want of anything better, however, supplementary foodstuffs can also be
distributed to make up for the deficiencies and imbalances in very young
herbage that has received excessive dressings of fertilizer.
Some of the common supplements will be discussed below.
 Fibrous foodstuffs at the beginning of the grazing season

The commencement of the grazing season is an extremely dangerous period
for tetany in the case of animals that have spent the winter indoors. It
would be wise to wait until the first of the grass has reached a height of
5-6 in. [12-15 cm.]. But winter keep is scarce and expensive, which means
that, as soon as the grass has begun to grow a little, the farmer is
compelled to put his stock out to graze. The problem. therefore, is to
take the necessary steps to minimize as far as possible the risks of
grazing this first growth of grass. The rules to be observed in this initial
period at grass have already been outlined. During the transitional
period and for a few days subsequently (say twelve days or
so) 2
it is advisable to compensate for the lack of crude fibre and carbohydrates
in the very young
grass 3
by feeding the animals hay, straw and cereal grains such as oats, barley,
etc.
 Buccal administration of magnesium

It has been seen that the risks of tetany appear to be very much greater
when the magnesium content of the herbage is relatively low. It was
logical, therefore, to feed supplements of magnesium in an attempt to
afford protection against hypomagnesaemia, an attempt all the more normal,
as supplements of magnesium salt fed to cows that were hypomagnesaemic but
not suffering from tetany had been observed (Fig. 13) to clearly and
rapidly raise the level of magnesium in the blood serum. This effect,
unfortunately, persists only during the period of the buccal administration,
and the magnesium is immediately "lost" in the urine. The organism,
moreover, has hardly any mobilizable reserves of magnesium. It is
understandable, therefore, that the administration (during a relatively
short time) of magnesium supplements in the ration will not allow the
animal to build up the magnesium reserves that would make it better able
to resist the hypomagnesaemic effects of the herbage. In other words,
preliminary supplements of magnesium, once they have been stopped, do not
continue to afford protection against the factors causing hypomagnesaemia,
should these factors continue to be present.
The most common and cheapest supplement of magnesium is magnesium oxide
(MgO),4
the recommended dose of which is 50-60 gm./day for a cow and 6-8
gm./day for a ewe. Distribution should commence two days before the
animals go out to grass, and continue through the transitional grazing
period (5-6 days) and for a fortnight subsequently. Even if given over
periods of several months, these doses will have no harmful secondary
effects.5
The magnesium oxide supplement can be mixed with wet beet pulp. If none is
available it can be given with crushed oats, but the beasts will sometimes
refuse it because of its bitter taste. Concentrate cubes are now available
containing magnesium, and cows will eat these without any trouble.
In Holland, in 1960, 330,000 cows received concentrated foodstuffs of this
nature supplying 50 gm. magnesium oxide (MgO) per head per day in the
ration. The consequent diminution in tetany cases was estimated at 70-80%.
If these results are correct this would appear to be a very efficient
method of "protection".
Other supplements of magnesium salts are also possible: for example,
magnesium sulphate, but this unfortunately presents the risk of
scouring.6
Magnesium bicarbonate has also proved effective (Fig. 13).
Administration of such supplements to animals at grass is obviously easy
in practice only in the case of milch cows being milked under cover. The
simplest method of supplementing the diet of a cow or ewe at pasture with
magnesium remains, therefore, as has been repeated again and again, to
supplement the herbage itself, increasing its magnesium content by applying
the necessary dressings of magnesium fertilizer. This is all the more to
be recommended, as these dressings of magnesium fertilizer are known to
alter not only the magnesium content of the herbage but also its
organic
composition.7

Lack of uniformity in results obtained from the buccal administration of
magnesium

Despite the success of supplementary magnesium administration, irregular
results have sometimes been
obtained.8
In the first instance it seems obvious that the mechanisms of magnesium
metabolism can be so profoundly upset that the organism is incapable of
using efficiently the exogenous magnesium supplied to it. It sometimes
happens even that the magnesium balance remains negative despite
magnesium supplementation.
ALLCROFT, for example, administered a daily supplement of 56 gm.
magnesium (MgO) to each cow on three farms when they were put out to grass.
The results on two farms were favourable, but on the third farm the
treatment had no effect. The "dietary record" of the herd on the third
farm might explain why it was incapable of utilizing magnesium supplements
efficiently.
 Supplements of sodium as a protection against grass tetany

As long ago as 1930 the Dutch pioneer SJOLLEMA expressed the opinion that
sodium deficiency played a decisive role in grass tetany and claimed to
have got protection against the disease by administering sodium.
Since then, practical men and scientists alike have obtained the most
contradictory results. In the long run, however, it was to appear as if
sodium does indeed play a decisive part in grass
tetany.9
It was noted, moreover, that cows on tetany pastures experienced a great
"salt hunger",10
and this logically led to their being fed a sodium supplement.
An average dairy cow has been seen to require 80 gm. salt per day. On
the other hand, it would seem wise to assume that grass supplies
practically no sodium at all, as is frequently the
case,11
and take the precaution of feeding a daily salt supplement of about 80 gm.
per cow (and 10 gm. per ewe). As in the case of magnesium, it is only easy
to distribute salt supplements to milch cows that are being milked under
cover. The old method of making salt licks available is still to be
recommended, therefore.
Another method consists of adding salt to the drinking-water. FRENS
advocates that water containing 2-5% salt (NaCl) should always be made
available to animals grazing young grass, the mineral composition of which
is unbalanced due to dressings of potassium fertilizer or liquid manure.
Another old forgotten method is to "salt" hay.
The most effective measure, however, is still to increase the sodium
content of the herbage by applying the required amounts of sodium to the
soil via the different sodium fertilizers.
 Contradictory results

As has just been said, the effectiveness of sodium supplements is much
disputed. Many investigators have voiced the opinion that, although this
supplement might be justified in other
circumstances,12
it is not effective against tetany.
On the other hand, a practical farmer in England, PICKERING, reports that
the addition of salt to the
ration 13
he was feeding to sheep greatly reduced the incidence of hypomagnesaemia
in the flock. More recently another English farmer, Rex PATERSON, who is
responsible for 40 herds of 65 cows seriously affected by tetany,
distributed salt supplements to two of the affected herds in which the
cows were not only extremely nervous and hyper-irritable but exhibited a
tendency towards "licking". He thought the results he obtained were
dramatic: the nervousness disappeared and there was an immediate increase
in the output of milk. And a point of fundamental importance: no
subsequent cases of tetany were observed. A slight
increase 14
in the mean magnesium content of the blood
serum 15
was also noted. In Germany BECKER found that distribution of 50 gm. salt
per cow per day helped to eliminate grass tetany.
These contradictory results are not surprising for, as has been
seen, sodium deficiency makes its pathological effects slowly felt.
Conversely, supplementary feeding is slow to take effect on the magnesium
metabolism of the cow. It is not a case, therefore, of administering
sodium supplements as a therapeutic, but of seeing that sodium plays its
part as a protector. To this end care must be taken that the ration always
contains a sufficiency of the element, supplied either by sodium
supplements or by the required applications of sodium fertilizer to the
herbage. This is all the more essential, as sodium deficiency, in the
long run, may have so seriously impaired the adrenal cortex that no
sodium supplement will allow it to resume its normal functions.
| Go To Chapter 38 |
| Grass Tetany Table of Contents |
| Home |
Ag. Library |
Health Library |
Sovereignty Library |
Notes
[Click on asterisk (*) at the end of a note to return
to the point you left in the text]
-
Or to arrange for older tufts of grass side by side with the very young
herbage in the pasture. *
-
This time depends on many factors: external temperature, spreading of
magnesium fertilizers, tendency towards tetany of the pasture in previous
years, etc. *
-
For the consequences of this deficiency of crude fibre and carbohydrates
on the health of the animal, see Chapter 25. *
-
Different qualities are available. The commercial products can differ in
quality, and therefore in potency, according to the primary substance
utilized and the method of preparation adopted (calcination temperature,
etc.). The light powder used in human medicine (magnesia usia) and obtained
by calcination of magnesium hydroxide is twenty times more soluble at a
pH of 6-6 (in the rumen) than cheap, heavy forms of magnesia obtained by
the calcination of different magnesium salts, such as the carbonates. The
light powder, however, is very expensive, and so it happens that it is
the less potent, heavy magnesia that is introduced into certain
concentrated foodstuffs.
In the U.S.A. there is an ultra-light quality of magnesia known as
"neoprene grade". Its reactive area is twenty times greater than that of
the so-called heavy magnesia. Consequently, it takes effect more rapidly
when administered buccally. *
-
In the U.S.A. more than 100 gm. magnesium (MgO) have been administered
per cow per day, but in several daily doses. *
-
HORVATH, in the U.S.A., reports having used it without any disadvantages.
*
-
See Soil, Gross and Cancer, Chap. 2, p. 5, and Table 11, p. 22. *
-
As in the case of parenteral magnesium therapy. *
-
Note also the sodium content of herbage and the influence of sodium
fertilizers. An outline has also been given of the influence of
sodium deficiency on the adrenal glands and the consequences thereof for
magnesium metabolism. *
-
Among others, PATERSON has reported the tremendous desire for salt
exhibited by cows in herds suffering from tetany. *
-
See, for example, Tables 11 and 26 and Chapter 26. *
-
Ie. in the case of other diseases caused by sodium deficiency:
sterility, bone fractures etc., not to mention reduced milk production.
*
-
Bone meal. *
-
In one of the herds this mean rose from 0-80 to 1-09 mg./100 c.c. and
in the other from 1-50 to 1-56 mg./100 c.c. The change in the external
symptoms, therefore, was apparently more marked than the rise in
the magnesium content of the blood serum. *
-
EVELETH reported that, in general, administration of bicarbonate of
soda increases the magnesium content of the blood serum. *
|
 |